By S. Harvey Mudd (auth.), Harold H. Draper (eds.)
Volume four of Advances in dietary study displays the elevated value that lately has been hooked up to food in lots of fields of scientific medication. This heightened curiosity in nutrients stems from the demonstration that the consumption of particular nutrition could have far-reaching outcomes, not just for regular metabolism, but in addition for metabolic procedures affecting medical or subclinical sickness. Conversely, many ailment states were proven to have formerly unrecognized results on nutrient functionality and metabolism. as well as subject matters of noticeable relevance to human scientific food, this quantity includes chapters facing the food of cells grown in tradition and of species which could supply insights into dietary problems of guy. To gether with its predecessors, quantity four offers graduate scholars and estab lished investigators with authoritative money owed of the prestige of study on a variety of subject matters of present curiosity in experimental and medical food. vii Contents bankruptcy 1. Vitamin-Responsive Genetic Abnormalities .......... . S. Harvey Mudd 1. Introduction.............................................. 1 2. old standpoint ...................................... 2 three. Genetic Abnormalities presently identified to Be nutrition Responsive. three four. Mechanisms Underlying nutrition Responsiveness. . . . . . . . . . . . . . . . 7 4.1. Defects within the Processing of a diet sooner than Its interplay in Cofactor shape with a specific Apoenzyme ........ 7 4.2. Defects particularly Apoenzymes that have interaction with the Cofactor types of supplementations. . . . . . . . . . . . . . . . . . sixteen . . . . . .
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Extra info for Advances in Nutritional Research
91:427. Haurani, F. , Hall, C. , 1979, Megaloblastic anemia as a result of an abnormal transcobalamin II (Cardeza), J. Clin, Invest. 64: 1253. 30 S. Harvey Mudd Hillman, R. , Keating, J. , and Williams, J. , 1978, Biotin-responsive proprionic acidemia presenting as the rumination syndrome, 1. Pediatr. 92:439. Hitzig, W. , Pluss, H. , 1974, Hereditary transcobalamin II deficiency: Clinical findings in a new family, 1. Pediatr. 85:622. Hommes, F. , Polman, H. , and Reerink, J. , 1968, Leigh's encephalomyelopathy: An inborn error of gluconeogenesis, Arch.
Freeman, J. , Finkelstein, J. , and Mudd, S. " A defect in methylation due to deficient 5, lO-methylenetetrahydrofolate reductase activity, N. Engl. J. Med. 292:491. Frimpter, G. , 1965, Cystathioninuria: Nature of the defect, Science 149:1095. Frimpter, G. , 1963, Cystathionuria, N. Engl. J. Med. 268:333. Frimpter, G. , Andelman, R. , and George, W. -dependency syndromes. New horizons in nutrition, Am. J. Clin. Nutr. 22:794. , 1965, Pyridoxine-responsive anaemias, Br. J. Haematol. 11:86. Gibbs, D.
Thus, there are good correlations between the presence of a small residual activity of cystathionine f3-synthase and Bs responsiveness on the one hand, and between the lack of residual activity and Bs nonresponsiveness on the other. With the advent of the use of the fibroblast system, liver biopsy for study of cystathionine f3-synthase deficient patients has largely ceased. Nevertheless, the results available from assays of hepatic cystathionine f3-synthase done by adequately sensitive methods confirm the correlation between Bs responsiveness and the presence of residual cystathionine f3-synthase activity.